Heart Failure Chymase Inhibition Prevents Fibronectin and Myofibrillar Loss and Improves Cardiomyocyte Function and LV Torsion Angle in Dogs With Isolated Mitral Regurgitation

نویسندگان

  • Betty Pat
  • Yuanwen Chen
  • Cheryl Killingsworth
  • Junying Zheng
  • Mustafa I. Ahmed
  • Ravi Desai
  • Henk Granzier
چکیده

Betty Pat, PhD; Yuanwen Chen, MD, PhD; Cheryl Killingsworth, DVM, PhD; James D. Gladden, BS; Ke Shi, MD; Junying Zheng, PhD; Pamela C. Powell, MS; Greg Walcott, MD, PhD; Mustafa I. Ahmed, MD; Himanshu Gupta, MD; Ravi Desai, MD; Chih-Chang Wei, PhD; Naoki Hase, MS; Tsunefumi Kobayashi, PhD; Abdelkarim Sabri, PhD; Henk Granzier, PhD; Thomas Denney, PhD; Michael Tillson, DVM; A. Ray Dillon, DVM; Ahsan Husain, PhD; Louis J. Dell’Italia, MD

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Chymase inhibition prevents fibronectin and myofibrillar loss and improves cardiomyocyte function and LV torsion angle in dogs with isolated mitral regurgitation.

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Chymase inhibition prevents cardiac fibrosis and improves diastolic dysfunction in the progression of heart failure.

BACKGROUND Angiotensin (Ang) II, which plays a crucial role in the cardiac remodeling process, is generated via angiotensin-converting enzyme (ACE); however, an alternative generation pathway, chymase, which is stored in the mast cells, also exists in the heart. Cardiac chymase is insensitive to ACE inhibitors (ACEIs), and heart chymase promotes interstitial fibrosis by affecting collagen metab...

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تاریخ انتشار 2010